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Certain tumors escape ferroptosis by upregulating system xc (a cystine/glutamate antiporter composed of SLC7A11 and SLC3A2) or glutathione peroxidase 4 (GPX4), a key enzyme that reduces lipid hydroperoxides and protects cells from ferroptotic death [1]
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It functions as a gene expression regulator and tissue repair stimulator, affecting over 4,000 human genes across pathways governing collagen synthesis, inflammation, wound healing, and cellular regeneration
Hydrogen peroxide (H 2 O 2, a strong oxidant) or Dithiothreitol (DTT, a cell permeable thiol reducing agent) were added after a stable baseline was reached at 30 s
BPC-157 studies demonstrated accelerated healing in Achilles tendon transection models[7], while TB-500 showed enhanced wound healing effects in various injury models[8]
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